MYO6 and hypertrophic cardiomyopathy: Loss of these functions underlies a number of phenotypes observed in the MYO6‐null Snell's waltzer mouse, or in humans harbouring mutations in the MYO6 gene, including deafness, astrogliosis, proteinuria as well as hypertrophic cardiomyopathy 2, 3, 4, 5, 6.