The BCL11A and ERC1 genes have been found to be disrupted by deletions at 2p16.1 and 12p13.33, respectively in children with CAS or with broader problems that also involve intellectual disability, motor difficulties, developmental problems (for BCL11A deletions) and psychiatric manifestations (for ERC1 deletions) [8, 9]. This evidence concerns the gene ERC1 and Intellectual disability.