A similar mechanism of OC-mediated escape from dormancy has been reported in breast-cancer cells that aberrantly express the vascular cell-adhesion molecule 1 (VCAM-1), recruit α4β1 OC precursors by engaging in VCAM-1/α4β1 binding to increase Oct activity, and stimulate tumor growth [166]. The gene discussed is BGLAP; the disease is neoplasm.