GLO1 and metabolic dysfunction-associated steatohepatitis: Taken together with previous reports of decreased hepatic formation of S-D-lactoylglutathione [21] and increased MG-H1 residue content of hepatic plasma protein [43] in experimental models of NASH, this finding suggested that increased GLO1 proteolysis in the liver in obese subjects leads to the increased dicarbonyl glycation implicated in NASH.