In a stenosis model of DVT, a cocktail of cytokines typically released by myelomonocytic cells including IL-12 and IL-18 fosters Ifnγ-production by TEM cells, and deficiency of these Ifnγ-producing cells reduces infiltration of lymphocyte antigen 6 complex locus C1 (Ly6C)hi inflammatory monocytes into thrombotic veins and improves thrombus resolution6. Here, IFNG is linked to deep vein thrombosis.