Indeed, in LNCaP-JAK1 cells, IFNγ activated STAT1 but not STAT3 (Figure S3A in Supplementary Material), abrogated NS3 production in EHDV-TAU-infected cells (Figure S3B in Supplementary Material), and supported oncolysis by non-productive viral infection (Figure S3C in Supplementary Material). The gene discussed is IFNG; the disease is viral infectious disease.