The results in the Table S1 in Supplementary Material showed that in omental and subcutaneous adipose tissues, insulin resistance inhibits the expression of Treg (forkhead family of transcription factor O1, FoxO1) and Th22 (ligand-activated helix-loop-helix transcription factor and aryl hydrocarbon receptor, AHR) regulators but promotes the expression of other Th subset regulators in visceral adipose tissues. The gene discussed is AHR; the disease is Insulin resistance.