Together, these findings suggest that glucose utilization was enhanced in all three subtypes (particularly in basal-like and HER2-enriched tissues) compared to luminal A breast cancer and are in agreement with evidence in the literature demonstrating that uptake of the glucose analogue fluordeoxyglucose F 18 (18F-FDG) in breast cancer patients correlates with their tumor proliferative potential. The gene discussed is ERBB2; the disease is neoplasm.