In NK cell-depleted mice, Ptgs1/Ptgs2−/− BRAFV600E tumors expressing XCL1 grew more slowly than mock-transduced cells (Figure 5F), suggesting that XCL1-mediated recruitment of cDC1 can partially compensate for the loss of tumor immune control caused by NK cell ablation. The gene discussed is MPPE1; the disease is neoplasm.