An oncogenic function was suggested in SOCS1 knockout mice, which developed sporadic CRC with strong tumor cell‐intrinsic STAT1 activation (Hanada et al., 2006), but otherwise, ApcMin‐induced intestinal tumorigenesis was not affected in STAT1 knockout mice (Liddle and Frank, 2008). The gene discussed is STAT1; the disease is colorectal carcinoma.