IL17A and experimental autoimmune encephalomyelitis: Th17 cells and IL‐17 play a prominent role in the development of TLSs during experimental autoimmune encephalomyelitis,35 but are not central to lymphoid neogenesis in inflamed salivary glands.38 Our data illustrates that while GC development in gp130F/F mice is linked with the heightened expression of the Th17‐associated markers Il17a, Il23 and Rorγt,30 the signature Th17 cytokine IL‐17 is not required for gastric tumourigenesis or the development of submucosal TLSs.