In addition, we examined the scar 1 week later, at day 28 after MI, and determined that iRhom2-deficient mice retained a significant number of CD68+ mononuclear cells within a disorganized matrix (Figure 4), suggestive of persistent inflammation and continued remodeling of the collagen and fibrous support during late-stage repair. This evidence concerns the gene CD68 and myocardial infarction.