In line with this, we found higher expression of SPRY4 than that of SPRY4-IT1 in three of the TGCT subtypes, whereas, in teratoma, the expression of SPRY4-IT1 was higher than that of SPRY4. The higher levels of the host gene SPRY4 and its intronic lncRNA SPRY4-IT1 with variable expression patterns in different TGCT subtypes suggest they are not simply oncogenes but rather have some independent regulatory mechanisms. The gene discussed is HAUS3; the disease is teratoma.