Since strong loss-of-function CrebA mutants are embryonic lethal, we tested the role of CrebA in response to infection by knocking it down in the fat body of adult flies using 3 independent RNAi constructs expressed under the control of the c564-Gal4 driver (Gal80ts; c564-Gal4 > UAS-CrebA-IR) and, separately, the Lpp-Gal4 driver (Gal80ts; Lpp-Gal4 > UAS-CrebA-IR) [36]. The gene discussed is LGALS4; the disease is infection.