Given the mixed effects exogenous oxytocin exposure (e.g., labor induction, labor augmentation, and/or postpartum hemorrhage prevention) has been shown to have on breastfeeding [44] and its potential long-term effect on endogenous levels [45], subsequent studies should measure exposure to account for potential differential levels by breastfeeding status and maternal-infant bonding. The gene discussed is OXT; the disease is postpartum hemorrhage.