IFNG and vasculitis: In the same study, we report an early increase of NK IFN-γ+ ex vivo, suggesting a cytokine milieu, rich of IL12, derived from dendritic cells (DC), and IFN-γ from NK, that could favor an increase of type I NKT producing IL17 that could be responsible of vasculitis, a pathological feature not only during Rickettsia spp.