Another study performed a deep sequencing analysis of mucinous ovarian tumors, showing a comparable pattern of recurrently mutated driver genes, including KRAS, CDKN2A, PIK3CA and PTEN, except for TP53 much more frequently mutated in carcinoma (57%) than in borderline (11.5%) tumors ([116], Figure 2). Here, KRAS is linked to carcinoma.