MAPK1 and ovarian carcinoma: Particularly, it was shown that miR-200a targets p38 and, through this mechanism, controls oxidative stress response: ovarian cancer patients with low miR-200a expression had poor prognosis compared with patients with high miR-200a expression [287]; miR-200c was found to be deregulated among stage I epithelial ovarian cancer patients who relapsed, compared with non-relapsers and was associated with patient survival [288].