At p32, a time point when the rat OIR model naturally undergoes regression of IVNV and ongoing vascularization of the peripheral avascular retina, there was no difference in AVA or IVNV between L-lucifshRNA control and either L-VEGFAshRNA or L-VEGF164shRNA (Fig. 2), providing evidence that Müller cell specific knockdown of VEGFA or VEGF164 did not lead to recurrent retinopathy. This evidence concerns the gene VEGFA and retinal disorder.