HIF1A and pancreatic ductal adenocarcinoma: The polymorphism in the miR-199a target site in HIF1A sequence is associated with pancreatic ductal adenocarcinoma risk [122], whereas forced overexpression of miR-199a in endometrial stromal cells (ESCs) attenuated HIF1A’s angiogenic potential during hypoxia via targeting both VEGFA and HIF1-α in these cells [123].