The results showed that the MyD88 protein expression levels normalized to TLR4 in FCD lesion were higher than that in peri-FCD (8.151 ± 1.042 vs. 4.528 ± 1.212; n = 8, P < 0.01; Fig. 1c), suggesting that MyD88 was recruited and formed TLR4/MyD88 complex in FCD lesion. Here, MYD88 is linked to fleck corneal dystrophy.