MFN1 and benign prostatic hyperplasia: Based on this, we speculated that the increased expression of Mfn1 in prostate promoted mitochondrial fusion, altered mitochondrial morphology, inhibited apoptosis in the occurrence and development of BPH; After administrated with rape bee pollen, plant source miR5338 was enriched in the prostate, combined to Mfn1 mRNA, reduced the expression level of Mfn1, inhibited mitochondrial fusion, reversed mitochondrial morphology, promoted cell apoptosis, improved BPH.