In silico analysis showed that BCL2 3’UTR contains a functional miR-204-binding site and an experiment with mutated BCL2 3’UTR showed confirmed that miR-204 targets Bcl-2 and its downregulation results in aberrant Bcl-2 expression in a gastric adenocarcinoma cell line [16]. This evidence concerns the gene BCL2 and gastric adenocarcinoma.