Although the adherens junction protein nectin-4 (NECTIN4/PVRL4), which is a recently identified epithelial receptor for MV [45,46], was not found to be regulated by the identified set of miRNAs, it is likely that adherens junction proteins, extracellular matrix-receptor interactions (highlighted pathway in Table 3), and the adhesion and regulation of the cytoskeleton are important biological processes involved in MV infection and measles immunity. The gene discussed is NECTIN4; the disease is measles.