CAV1 and schizophrenia: Other potential gene linkages in this locus (NRCAM and ST7) are relatively weak, indicating that the linkage must be accounted for, at least in part, by other Auts9 genes, potentially partially by Cav1. CAV1 has also been implicated as a potential target for schizophrenia therapy due to its interaction with DISC1, and its ability to modulate DISC1 expression in neurons (Kassan et al., 2017).