TP53 and neoplasm: Inhibition of MK2 using small molecule tool compounds has also been shown to synergize with blockade of other cell cycle checkpoint kinases in Ras‐driven solid tumors, augmenting tumor cell killing in a p53‐dependent manner.53 The discovery of safe and specific small molecule inhibitors of MK2 for rheumatoid arthritis54 therapy is also an active area of industrial research.