In GC cells, lncRNA H19 could be processed into miR-675, which subsequently activates protein kinase B/mammalian target of rapamycin (Akt/mTOR) pathway as well as enhances the invasion of GC cells; therefore, the function of H19 is partially dependent on its downstream product miR-675 [94]. The gene discussed is AKT1; the disease is gastric cancer.