In line with this, transplantation of the bone marrow deficient for CCL3 (a ligand for CCR1, CCR4 and CCR5) to LDLR‐deficient mice resulted in diminished aortic atherosclerosis progression likely due to prevention of intraplaque accumulation CCL3‐deficient neutrophils. This evidence concerns the gene CCL3 and aortic atherosclerosis.