Corresponding to a previous report that IL-8 is a transcription activation target of RAS signaling14, our transcriptome analysis revealed that FGFR3 kinase-dependent activation of MAPK signaling would promote not only cell proliferation but also an inflammatory response15 in FGFR3-TACC3 fusion-positive cervical cancer (Fig. 3f). This evidence concerns the gene FGFR3 and cervical carcinoma.