To examine the likelihood of TrLp-evoked repolarization of pro-tumor ARG1high/iNOSlow-M2-like TAM into anti-tumor ARG1low/iNOShigh-M1-like Iba1(+) TAM, we performed flow cytometry analysis of immunostained cells dissociated from vehicle-, CLp(−) and TrLp-treated GBM tumors (Figure 5) [14]. The gene discussed is AIF1; the disease is neoplasm.