The anti-allergy effects of acteoside were due to downregulation of the expressions of the chemokine ligand (CCL) 1, CCL2, CCL3, CCL4, Fc fragment of IgE, high affinity I, receptor for alpha polypeptide (FCER1A), nuclear factor of activated T cell, cytoplasmic, and calcineurin-dependent 1 (NFATC1) genes and inhibition of the mitogen-activated protein kinase (MAPK) pathway through decreased C-jun N terminal kinase (JNK) phosphorylation [111]. The gene discussed is NFATC1; the disease is allergic disease.