MKI67 and acute myeloid leukemia: The growth-inhibitory response to TGFβ1 noted in TF-1a cells compared to KG-1a and Kasumi-3 cells and the pronounced loss of Ki-67 in TF-1a after TGFβ1 treatment compared to Kasumi-3 and M0-91 cells supported its selection to model and characterize AML cell dormancy in the study.