MCL1 and gastric cancer: Previous studies revealed that MCL1 was inversely correlated with the level of some miRNAs such as miR-193b [32] and miR-125b [3], and MCL1 expression could induce epithelial–mesenchymal transition (EMT) via some signaling pathways, which subsequently stimulated the invasive and migratory capacity of human GC cells [4].