In univariable analyses, participants with lobar intracerebral haemorrhage and moderate or severe CAA were significantly more likely to be APOE ɛ4 carriers, and to have a strictly lobar intracerebral haemorrhage, subarachnoid haemorrhage, and finger-like projections from the intracerebral haemorrhage than participants with lobar intracerebral haemorrhage and absent or mild CAA (table 1). This evidence concerns the gene APOE and subarachnoid hemorrhage.