Chronic myeloid leukemia (CML) is a form of hematopoietic stem cell disease characterized by the presence of the oncogene BCR-ABL, which is created by the fusion of BCR and ABL genes and results in a constitutively active BCR-ABL tyrosine kinase protein [1]. This evidence concerns the gene ABL1 and chronic myelogenous leukemia, BCR-ABL1 positive.