These findings suggest potential transcriptional interactions involving FOXG1 and TLE1 with other factors previously implicated in GBM, including CEBPB, CTCF and JUND (Ayala‐Ortega et al., 2016; Rong et al., 2009; Talasila et al., 2017). The gene discussed is JUND; the disease is glioblastoma.