IL17A and infection: 2006). However, our findings are more in agreement with those of the authors who have suggested that neutrophils are not sufficient to control the infection despite their recruitment to the infection site (Massey et al. 2014). In addition, Massey et al. (2014) found an important increase in IL‐17 in infected animals compared with non‐infected control animals. This cytokine is recognized as the main mechanism whereby Th17 cells contribute to tissue damage due to prolonged neutrophil recruitment (Fogli et al. 2013).