It was noteworthy that NTCP deficiency might not be the unique causative factor for indirect hyperbilirubinemia in the two infants, since the total bile acid levels kept beyond the upper limit of the reference range while hyperbilirubinemia got alleviated or resolved gradually in both patients (Table 1). The gene discussed is SLC10A1; the disease is hyperinsulinemic hypoglycemia, familial, 4.