MERTK and gastric cancer: In the current study, we have found that 1) MerTK inhibitor treatment resulted in considerable inhibition of cell growth and downstream signaling; 2) MerTK phosphorylation, not total MerTK expression, is likely more predictive of therapeutic success; 3) p-MerTK protein overexpression by IHC was found in 18% (17/87) of GC patients; 4) RXDX-106 inhibited cell proliferation in MerTK activated gastric cancer cell line.