In conclusion, the zinc transporter SLC39A7, whose abundance is increased in cervical cancers, and knockdown of SLC39A7 mediate inhibition of cell proliferation, migration, invasion and induction of apoptosis though upregulation of Bax and E-cadherin and downregulation of Bcl-2 and MMP-2, supports a metastasis promoted role for SLC39A7 in cervical cancer. This evidence concerns the gene SLC39A7 and cervical carcinoma.