This cellular defect might also contribute to the impaired immune response to EBV infection in RASGRP1‐deficient patients since iNKT cells have the ability to control EBV‐infected B cells and are often defective in primary deficiencies characterized by high susceptibility to EBV (Chung et al, 2013; Veillette et al, 2013). Here, RASGRP1 is linked to Epstein-Barr virus infection.