AGT and cardiac arrest: The mechanisms surrounding a potential benefit of Ang II in cardiac arrest are only speculative but may include: (a) maintenance of adequate systemic BP to vital organs [60], (b) manipulation of intracellular calcium levels leading to increased inotropy [61, 62], (c) potentiation of catecholamines [63, 64], and (d) increased afterload for enhancement of coronary perfusion [65].