KRAS and neoplasm: Targeting glutamine synthetase in the stroma and glutaminase in the cancer cells blocked this metabolic symbiosis and reduced tumor growth, burden, and metastasis in a SKOV3 orthotopic tumor model.136 EVs secreted by patient‐derived prostate CAFs and pancreatic CAF‐19 line supported growth of pancreatic and prostate cancer cell lines via KRas‐independent mechanisms.