However, responses in the controls were distinguished by inflammation-associated pathways that are consistent with the pathogenesis of multiple sclerosis (ES = 7.978), hypercytokinemia and hyperchemokinemia in the pathogenesis of influenza (ES = 6.65), and NF-κB signaling (ES = 2.79), and these patterns were enhanced relative to that observed in the vaccinated animals (ES = 4.892, 2.051, and no significant enrichment; respectively). Here, NFKB1 is linked to influenza.