Some reports underscored the capacity of FoxM1, in GBM, to bind β-catenin in the cytoplasm and translocate it to the nucleus in a Wnt-independent fashion [109,110], increasing the expression level of c-Myc and cyclin D1 genes [110], as well as of Myb-related protein B (MYBL2) [111]. The gene discussed is MYC; the disease is glioblastoma.