Keratinocyte-derived cytokines such as TSLP [65], which is known to promote the AD-like phenotype [66], IL-25, IL-33, and granulocyte-macrophage colony-stimulating factor (GM-CSF) influence innate lymphoid cells (ILCs) and increase the production of Th2 chemokines: CCL17 (thymus- and activation-regulated chemokine (TARC)), CCL22, and an eosinophil chemoattractant: CCL5 (regulated upon activation, normal T cell expressed and secreted (RANTES)) [67]. This evidence concerns the gene IL33 and Alzheimer disease.