NAGK and idiopathic inflammatory myopathy: These activities include enhanced kinesin associated intracellular transport [70] that is associated with activation of immune cells in idiopathic inflammatory myopathies [71], duplication of genomic DNA by DNA replication licensing factors [72], regulatory roles of N-acetyl-D-glucosamine kinase in gene expression [73], meiosis cytokinesis by brunelleschi [74], and nuclear import of splicing factors through nuclear pore complexes by transportin [75] and importin-13 [76].