As described [15], STm infection induced an early autophagic response in WT DCs and MΦs, as shown by induction of the active lipidated form of LC3 (LC3-II) relative to the unlipidated LC3-I form in cell lysates following infection (Figs5D, 5E, S6F and S6G). The gene discussed is MAP1LC3A; the disease is infection.