Surprisingly, (likely) pathogenic mutations were more frequently present in DCM- than in ARVC-cases (60% versus 40%) although the majority of currently known ARVC-genes with the exception of the minor disease genes TGFB3, CTNNA3 (see www.arvcdatabase.info), [4], FLNC [30], and SCN5A [31] were analyzed. Here, SCN5A is linked to familial dilated cardiomyopathy.