Although PCLP1 expressed on high endothelial venules interacts with L-selectin during lymphocyte recruitment [4], the failure of a blocking anti-L-selectin antibody to abrogate PCLP1-induced cell-to-cell adhesion in B-cell lymphoma point to another ligand as responsible for this interaction. This evidence concerns the gene SELL and B-cell non-Hodgkin lymphoma.