Total nevus density has been linked to sun exposure [13], lighter phototypes, a history of sunburns in children [14, 15], melanomas with contiguous neval remnants [16], familial cases of dysplastic nevus syndrome and melanoma [17], genetic cell-cycle control factors [18, 19] such as the CDKN2A gene [20, 21] and others [22], as well as an absent p53 expression within melanoma tumor samples, which supports the divergent pathway model [1, 23]. This evidence concerns the gene TP53 and melanoma.